Bibliography
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BACE1 Mediates HIV-Associated and Excitotoxic Neuronal Damage Through an APP-Dependent Mechanism. J Neurosci. 2018 ;38(18):4288-4300.
. Efavirenz Is Predicted To Accumulate in Brain Tissue: an In Silico, In Vitro, and In Vivo Investigation. Antimicrob Agents Chemother. 2017 ;61(1).
. HIV alters neuronal mitochondrial fission/fusion in the brain during HIV-associated neurocognitive disorders. Neurobiol Dis. 2016 ;86:154-69.
The HIV Protein gp120 Alters Mitochondrial Dynamics in Neurons. Neurotox Res. 2016 ;29(4):583-593.
E2F1 in neurons is cleaved by calpain in an NMDA receptor-dependent manner in a model of HIV-induced neurotoxicity. J Neurochem. 2015 ;132(6):742-55.
. Gp120 in the pathogenesis of human immunodeficiency virus-associated pain. Ann Neurol. 2014 ;75(6):837-50.
Neuronal ferritin heavy chain and drug abuse affect HIV-associated cognitive dysfunction. J Clin Invest. 2014 ;124(2):656-69.
Nonmuscle myosin light-chain kinase mediates microglial migration induced by HIV Tat: involvement of β1 integrins. FASEB J. 2013 ;27(4):1532-48.
. HIV-1 Tat-mediated induction of platelet-derived growth factor in astrocytes: role of early growth response gene 1. J Immunol. 2011 ;186(7):4119-29.
. Activation of cyclin-dependent kinase 5 by calpains contributes to human immunodeficiency virus-induced neurotoxicity. J Neurochem. 2007 ;103(2):439-55.
Human immunodeficiency virus-1/surface glycoprotein 120 induces apoptosis through RNA-activated protein kinase signaling in neurons. The Journal of Neuroscience: The Official Journal of the Society for Neuroscience [Internet]. 2007 ;27:11047-11055. Available from: http://www.ncbi.nlm.nih.gov/pubmed/17928446
. Tubulin-mediated binding of human immunodeficiency virus-1 Tat to the cytoskeleton causes proteasomal-dependent degradation of microtubule-associated protein 2 and neuronal damage. The Journal of Neuroscience: The Official Journal of the Society for Neuroscience [Internet]. 2006 ;26:4054-62. Available from: http://www.ncbi.nlm.nih.gov/pubmed/16611822
. Cardiomyocytes undergo apoptosis in human immunodeficiency virus cardiomyopathy through mitochondrion- and death receptor-controlled pathways. Proceedings of the National Academy of Sciences of the United States of America [Internet]. 2002 ;99(22):14386-91. Available from: http://www.ncbi.nlm.nih.gov/pubmed/12379743